Article Text
Abstract
We describe a case of truncal sensory polyneuropathy in a patient with light-chain amyloidosis. We highlight the clinical signs and differential diagnoses related to the presentation.
- NEUROPATHY
- AMYLOID
Data availability statement
Data sharing not applicable as no datasets generated and/or analysed for this study. Not applicable.
Statistics from Altmetric.com
Data availability statement
Data sharing not applicable as no datasets generated and/or analysed for this study. Not applicable.
Footnotes
Twitter @drantieclarke
Contributors AJC drafted the manuscript, sought patient consent and captured the patient photographs. ASC edited the manuscript and provided the neurophysiology results. CB edited the manuscript. GMH conceptualised the case and edited the manuscript.
Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.
Competing interests None declared.
Provenance and peer review Not commissioned. Externally peer reviewed by Mary Reilly, London, UK and Kathryn Brennan, Glasgow, UK.
Read the full text or download the PDF:
Other content recommended for you
- Paraproteinaemic neuropathy: MGUS and beyond
- Untangling a case of painful neuropathy
- Cardiac transthyretin amyloidosis
- Non-alcoholic fatty liver disease is associated with higher levels of objectively measured sedentary behaviour and lower levels of physical activity than matched healthy controls
- The spectrum of immune-mediated autonomic neuropathies: insights from the clinicopathological features
- A study of the neuropathy associated with transthyretin amyloidosis (ATTR) in the UK
- Amyloidosis
- Peripheral nerve diseases
- Transthyretin (ATTR) amyloidosis: clinical spectrum, molecular pathogenesis and disease-modifying treatments
- Lifestyle intervention for non-alcoholic fatty liver disease: prospective cohort study of its efficacy and factors related to improvement